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dc.contributor.authorLee, S.-
dc.contributor.authorLee, Y.S.-
dc.contributor.authorJung, S.H.-
dc.contributor.authorShin, K.H.-
dc.contributor.authorKim, B.-K.-
dc.date.accessioned2024-01-21T06:34:38Z-
dc.date.available2024-01-21T06:34:38Z-
dc.date.created2021-09-02-
dc.date.issued2004-09-
dc.identifier.issn0253-3073-
dc.identifier.urihttps://pubs.kist.re.kr/handle/201004/137295-
dc.description.abstractEffects of the EtOH extract from Pelvetia siliquosa on CCl 4-induced hepatotoxicity as well as streptozotocin-induced diabetes in rats were investigated. The EtOH extract was found to cause an inhibition of the rise in the transaminase activities in CCl4-intoxicated rats. Also, the EtOH extract exhibited a rat lens aldose reductase inhibition in vitro and showed an inhibition of not only glucose concentrations but also sorbitol accumulations in the lenses, red blood cells and sciatic nerves in the STZ-induced diabetic rats in vivo. These results suggested that this plant might possess hepatoprotective and anti-diabetic activities.-
dc.languageKorean-
dc.subjectalcohol-
dc.subjectaldehyde reductase-
dc.subjectaminotransferase-
dc.subjectcarbon tetrachloride-
dc.subjectepalrestat-
dc.subjectglucose-
dc.subjectliver enzyme-
dc.subjectmetformin-
dc.subjectpelvetia siliquosa extract-
dc.subjectplant extract-
dc.subjectsilymarin-
dc.subjectsorbitol-
dc.subjectunclassified drug-
dc.subjectanimal cell-
dc.subjectanimal model-
dc.subjectanimal tissue-
dc.subjectantidiabetic activity-
dc.subjectarticle-
dc.subjectcarbohydrate metabolism-
dc.subjectcontrolled study-
dc.subjectdrug activity-
dc.subjectdrug effect-
dc.subjectenzyme activity-
dc.subjectenzyme inhibition-
dc.subjecterythrocyte-
dc.subjectglucose metabolism-
dc.subjectlens-
dc.subjectliver protection-
dc.subjectliver toxicity-
dc.subjectmedicinal plant-
dc.subjectmetabolic regulation-
dc.subjectnonhuman-
dc.subjectPelevetia siliquosa-
dc.subjectrat-
dc.subjectsciatic nerve-
dc.subjectstreptozocin diabetes-
dc.titleEffects of the extracts from Pelvetia siliquosa on metabolism regulation in rats-
dc.typeArticle-
dc.description.journalClass1-
dc.identifier.bibliographicCitationKorean Journal of Pharmacognosy, v.35, no.3, pp.203 - 206-
dc.citation.titleKorean Journal of Pharmacognosy-
dc.citation.volume35-
dc.citation.number3-
dc.citation.startPage203-
dc.citation.endPage206-
dc.description.journalRegisteredClassscopus-
dc.description.journalRegisteredClasskci-
dc.description.journalRegisteredClassother-
dc.identifier.kciidART000940393-
dc.identifier.scopusid2-s2.0-5444251017-
dc.type.docTypeArticle-
dc.subject.keywordPlusalcohol-
dc.subject.keywordPlusaldehyde reductase-
dc.subject.keywordPlusaminotransferase-
dc.subject.keywordPluscarbon tetrachloride-
dc.subject.keywordPlusepalrestat-
dc.subject.keywordPlusglucose-
dc.subject.keywordPlusliver enzyme-
dc.subject.keywordPlusmetformin-
dc.subject.keywordPluspelvetia siliquosa extract-
dc.subject.keywordPlusplant extract-
dc.subject.keywordPlussilymarin-
dc.subject.keywordPlussorbitol-
dc.subject.keywordPlusunclassified drug-
dc.subject.keywordPlusanimal cell-
dc.subject.keywordPlusanimal model-
dc.subject.keywordPlusanimal tissue-
dc.subject.keywordPlusantidiabetic activity-
dc.subject.keywordPlusarticle-
dc.subject.keywordPluscarbohydrate metabolism-
dc.subject.keywordPluscontrolled study-
dc.subject.keywordPlusdrug activity-
dc.subject.keywordPlusdrug effect-
dc.subject.keywordPlusenzyme activity-
dc.subject.keywordPlusenzyme inhibition-
dc.subject.keywordPluserythrocyte-
dc.subject.keywordPlusglucose metabolism-
dc.subject.keywordPluslens-
dc.subject.keywordPlusliver protection-
dc.subject.keywordPlusliver toxicity-
dc.subject.keywordPlusmedicinal plant-
dc.subject.keywordPlusmetabolic regulation-
dc.subject.keywordPlusnonhuman-
dc.subject.keywordPlusPelevetia siliquosa-
dc.subject.keywordPlusrat-
dc.subject.keywordPlussciatic nerve-
dc.subject.keywordPlusstreptozocin diabetes-
dc.subject.keywordAuthorDiabetes-
dc.subject.keywordAuthorFucaceae-
dc.subject.keywordAuthorHepatotoxicity-
dc.subject.keywordAuthorPelvetia siliquosa-
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