Lee, Sung Ryul Kwak, Jong Hwan Kim, Hyoung Ja Pyo, Suhkneung 2024-01-21T01:04:35Z 2024-01-21T01:04:35Z 2021-09-02 2007-04-01 0960-894X https://pubs.kist.re.kr/handle/201004/134463 This study examined the neuroprotective effects of kobophenol A (kob A), oligomeric stillbene, and a resveratrol tetramer. Neuronal death induced by the withdrawal of tropic support was ameliorated by kob A. The protective effect of kob A against nitrosative/oxidative or mitochondrial damages resulted in the inhibition of the ROS, intracellular calcium ion level, and mitochondrial transmembrane potential changes on SH-SY5Y cells. (c) 2007 Elsevier Ltd. All rights reserved. English PERGAMON-ELSEVIER SCIENCE LTD NITRIC-OXIDE INDUCED APOPTOSIS OLIGOSTILBENES PARAQUAT Neuroprotective effects of kobophenol A against the withdrawal of tropic support, nitrosative stress, and mitochondrial damage in SH-SY5Y neuroblastoma cells Article 10.1016/j.bmcl.2007.01.078 1 BIOORGANIC & MEDICINAL CHEMISTRY LETTERS, v.17, no.7, pp.1879 - 1882 BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 17 7 1879 1882 scie scopus 000245827900009 2-s2.0-33847648508 Chemistry, Medicinal Chemistry, Organic Pharmacology & Pharmacy Chemistry Article NITRIC-OXIDE INDUCED APOPTOSIS OLIGOSTILBENES PARAQUAT kobophenol A SH-SY5Y neuroprotection